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Impaired insulin signaling, diminished mitochondrial oxidative capacity, lipid peroxidation, ferroptosis, and microbial dysbiosis not only exacerbate hepatic steatosis but also facilitate the progression from simple steatosis to more severe forms of the disease, such as NASH and cirrhosis [51,56,63,64,65,66,67]

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Thioredoxin can mediate denitrosylation of target proteins by removing the NO moiety, in turn, converting thioredoxin to the disulfide form in an NADPH-dependent reaction (Figure 3)

By combining them, the BPC-157 KPV stack may offer accelerated tissue healing, making it particularly relevant for studies focused on wound healing, tissue regeneration, and injury recovery