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2004), activate Nrf2 to inhibit the expression of BACE1 and BACE1-AS through the modification of the cysteine residue of Keap1 to prevent the production of A, a key pathogenic process in the early stages of AD (Dinkova-Kostova et al

In order to develop a more effective treatment strategy, as well as approaches to prevention, a critical analysis of the ways of immunotherapeutic use of dietary supplements of I, Se, Zn, Mg and other minerals in AITD was carried out

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