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Discussion The data presented here, together with previous publications 29,30 , show that a GSH-binding site is conserved across many enteroviruses

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S6K1-X was dominant negative for translation, explaining the majority of HNEylated NCBP1-induced translation inhibition and how NCBP1(C436)-specific HNEylation impaired translation

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Differences between wild-type and mutant nodules could be indicative of the involvement of (h)GSH at different stages of nodule development or in defense responses associated with ineffective symbiosis

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