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1 2 "Selenofolate inhibits the proliferation of IGROV1 cancer cells independently from folate receptor alpha"
doi: 10.1039/c9fo01014a Summary Keywords gastric cancer, oxidative stress, signaling pathways, pathophysiology, therapeutic targets Citation Liu Y, Shi Y, Han R, Liu C, Qin X, Li P and Gu R (2023) Signaling pathways of oxidative stress response: the potential therapeutic targets in gastric cancer

doi: 10.3109/15563650.2011.609821 [PubMed] 22998987 Hodgman MJ, Garrard AR

Secondly, it is known that the process of cell death is an extremely complicated interconnected process involving multiple molecules

(235), and its decreased content may cause higher oxidative stress in the thyroid (235)

At the same time, the absence of Gsr and TrxR in these cells results in an oxidized cytosol in which Grx1-disulfide or Grx-SSG accumulates
