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ROS are located upstream of many inflammatory signaling pathways, upregulate the expression of other inflammatory factors, interfere with signaling pathways that regulate growth factors, and often alter signal transduction

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No published research has demonstrated cancer promotion in any animal or human study

demonstrated that MSCs formed connexin 43-based gap junctional channels with the alveolar epithelium and released mitochondria-containing EVs, which are subsequently phagocytosed by epithelial cells [124]

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MIP-1, macrophage inflammatory protein 1-beta