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Six were depleted in the frail brain, including cytoskeletal proteoforms derived from MAP2, -tubulin and vimentin, whilst 16 were increased with cognitive frailty, including CHGA/B, CCK and other cytoskeletal peptides such as -tubulin and PALM (Fig

Karlsson, M., Contreras, J

certainly lends credence to the idea that NSI-189 is a 5-HT3-AGONIST...!) and others all cause psychosis - but they have such diverse and complex mechanisms, that one can't say it's because of 5-HT3-agonism, more like a combination of actions

JAMA Intern Med 179(5):633640

They are not law

While many regenerative therapies remain investigational, ongoing clinical research continues to expand our understanding of how biological treatments may support neurological development and quality of life for individuals with ASD