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However, even under inflammation-associated hypoxia, the remaining oxygen in the intestinal tissue is sufficient to support mitochondrial oxidative phosphorylation (OXPHOS) 15 , and these changes cannot explain the complex and early (before onset of histological inflammation) alterations in epithelial metabolism observed in intestinal inflammation 13,18,20

The disconnect between preclinical efficacy and human clinical outcomes remains unexplained and represents a significant limitation for potential therapeutic applications

Chen ZX, Chang M, Peng YL, Zhao L, Zhan YR, Wang LJ, et al

These parameters are illustrated schematically in Figure 32 where the volume of the beakers into which the drugs diffuse represents the volume of distribution, and the size of the outflow drain in Figures 32B and 32D represents the clearance

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doi: 10.1104/pp.97.3.863