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Nonetheless, when a cell (e.g., a hepatocyte) is exposed to inflammation or oxidative stress, NRF-2 has the capacity to escape from regulation by KEAP1 through the increase in the transcription of mRNA from NRF-2, with the objective of depleting the reserves of KEAP1, which cannot be recycled because of the suppression of another NRF-2 [21]

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Cuproptosis-associated genes (CAGs) contribute to the prognosis prediction and potential therapeutic targets in hepatocellular carcinoma

If intranasal bioavailability is 10% relative to subcutaneous, a 10-fold higher nasal dose would be needed to match exposure, but ceiling effects, tolerability, and mucosal volume limits complicate this further

Rev Port Cardiol 35(1):4157 Sousa T et al (2012) Lipid peroxidation and antioxidants in arterial hypertension

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