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C., Yin, M

AGEs are known to induce via oxidative stress several stress-induced transcription factors with the concomitant production of inflammatory cytokines

Toxicant channeling may provide an explanation for the finding that the S 3 segments of the proximal tubules are especially vulnerable to cysteine S -conjugates derived from halogenated alkenes

Endpoints tend to be tissue histology or molecular markers, rather than functional fertility

These differences can be used as intervention targets in future studies

In contrast, serine-induced mTOR inhibits Tregs suppressive activity (impeding Foxp3 expression), but this can be reversed by serine degradation to glutathione [110], reinforcing that the manner by which cells metabolize some amino acids can influence their function, being a target for therapies