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By incorporating miRNA structures into gene therapy agents, it aims to inhibit the production of the mutant Z-AAT protein and promote the production of functional A1AT ( These findings highlight the potential of A1AT augmentation therapy to modulate the inflammatory response in A1ATD, reduce neutrophil-mediated damage, and improve clinical outcomes in various manifestations of the disease, including lung inflammation and panniculitis

Its prevalence is inversely correlated with body weight, inflammation, metabolic syndrome, and both type 1 and type 2 diabetes

Disease-associated astrocytes in Alzheimer's disease and aging

Initiation and progression of -synuclein pathology in Parkinsons disease

[DOI] [PMC free article] [PubMed] [Google Scholar] 28.Rosenberg H.R., Culik R

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