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Additional file 3 : Table S3

As an autoimmune disease, the disturbance of the tolerance against self-antigens is the main pathogenic mechanism with a large cellular interplay that involves TFCs, stromal cells and lymphocytes

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By further elucidating the pathogenic mechanism by which hepatic iron accumulation triggers ferroptosis through the c-Myc-Acsl4 regulatory axis, thereby accelerating the progression of metabolic-associated steatohepatitis (MASH), our research team identified a novel iron chelator, FOT1 (FerroTerminator1, formerly known as CN128), through drug screening, which can prevent and treat the onset and progression of MASH disease [117]

3.6 C-PC Alone or Combined with CDDP Reduces ROS Production in GBM Cells We evaluated the effects of C-PC, CDDP, and their combination on ROS generation in GBM cells by measuring extracellular O2 and intracellular H2O2 levels in U87 and U87-EGFRvIII cells

Abbate, R