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The immune response in Graves disease is characterized by involvement of both Th2 and Th17 pathways, with interleukin (IL)-4, IL-6, and IL-17 contributing to B-cell activation and antibody production, while defects in immune checkpoints, such as impaired regulatory T-cell activity, promote loss of tolerance [17,18]

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To elucidate the contributions of hematopoietic versus endothelial cells to the control of TG excursion in Dpp4 EC/ mice, we reanalyzed these parameters after BMT

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RF microneedling involves using a device that combines microneedling with radiofrequency energy to penetrate the skin and stimulate fibroblastic function to increase collagen, elastic fiber, and hyaluronic acid production