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2014 studies [38, 50] were excluded as they reported results for relevant interventions with only secondary comparators

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In contrast, when GSDMC expression is upregulated, it becomes the preferred substrate of caspase-8, and its cleavage products directly trigger pyroptosis, thereby hijacking the classical apoptosis-inducing signal [300]

Based on these comprehensive research findings, we speculate that the regulation of the T1D immune system and inflammatory pathways may be closely linked to butyrate, a key metabolite produced by Clostridium

Increased oxidative stress and inflammation from these illnesses cause the body to use up glutathione more rapidly
doi: 10.1016/j.funbio.2024.02.005 397 KipkogeiKKiptuiKKipropE