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This elegant study provided evidence that platelets act as sensors of bacterial infection by demonstrating that activation of platelet TLR4 stimulates the binding of activated platelets to neutrophils, leading to the release of NETs to catch and kill bacteria

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Nociceptors: the sensors of the pain pathway

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Immunoblotting analyses were performed on n = 4 tissue samples from control group and n = 5 tissue samples from HnB group
