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Salidroside exerts neuroprotective properties by inhibiting A-induced ferroptosis in AD mouse neurons, and its mechanism is related to Nrf2 activation and the promotion of downstream NQO1, HO-1, and GPX4 protein expression
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PLR did not account for any indirect effect of ave PM 2.5 on VABS (Fig
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[29] The drug undergoes extensive hepatic metabolism, primarily through cytochrome P450 enzymes, particularly CYP3A4, CYP1A2, and CYP2D6
