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The convergence of Pb-induced QA elevation, astrocytic dysfunction, and EAAT3-mediated glutamate accumulation creates a vicious cycle of NMDA receptor overactivation, oxidative damage, and neuronal loss

Transition of Nano-Architectures Through Self-Assembly of Lipidated beta(3)-Tripeptide Foldamers

93,94,95,96 Advancements in carrier technology and nucleic acid modification techniques have led to significant progress in overcoming these challenges, as exemplified by the first siRNA drug, patisiran (Onpattro), which has been approved for treating hereditary transthyretin-mediated amyloidosis by degrading mRNA encoding transthyretin (TTR)

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internalization of the glutamate transporter in response to AMPH leads to enhanced glutamatergic responses in DA neurons [4]

Farnesoid X receptor represses hepatic lipase gene expression