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Consistent with this, adenoviral overexpression of PCSK1 in -cells increases islet GLP-1 production and secretion, ultimately leading to enhanced glucose-stimulation of insulin secretion and improved survival of the islets [165]

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Glucagon is usually thought of as a glucose- raising hormone, but in the context of simultaneous GLP-1/GIP agonism the net effect studied in trials was increased energy expenditure alongside strong glycemic control a combination that appears to explain the outsized weight effect observed in the research