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PRP has been shown to stimulate collagen production and reduce degenerative changes at the cellular level, directly addressing the underlying pathology of chronic tendinopathy (PMID: 29524997)

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Research in Pharmaceuticals (MDPI, 2025) reviews BPC-157's mechanisms across tissue injury and repair models, documenting its effects via VEGFR2-Akt-eNOS angiogenic pathways, nitric oxide signaling, and growth hormone receptor expression upregulation in tendon fibroblasts

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