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studied the effect of the enhanced GPx1 expression against hydroperoxide-, doxorubicin-, and radiation-induced toxicity in the human breast adenocarcinoma cell line (MCF7) [52]

The APE1 inefficiently cleaves but recruits various transcription factors to activate downstream genes, such as vascular endothelial growth factor (VEGF) and endonuclease IIIlike protein 1 (NTHL1) [95]

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This combination therapy not only effectively disrupts the metabolic adaptation caused by TP53 mutations but also provides a new direction for the treatment of CRPC and other prostate cancers by targeting key metabolic pathways