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2b the data was inadvertently shifted upwards and has now been corrected in the HTML and PDF versions of the article
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The pathogenesis of MS is attributed to mitochondrial damage and subsequent loss of energy in the CNS of susceptible individuals by oxidative stress, as evidenced by investigations [110,111,112]
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These effects were not reproduced in 3-AR knockout mice, suggesting that the 3-AR plays a key role in the compounds chronic metabolic effects (Heffernan et al
