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GSH then directs the migration of inflammatory polymorphonuclear neutrophils away from the lung, where they cause ARDS, and toward the site of infection, where they kill microorganisms

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endowed FTn with spatially controlled mosaic surface polyethylene glycol (PEG) coatings, facilitating FTns deep penetration through the tumor extracellular matrix (ECM) to reach hypoxic tumor tissues, providing significant therapeutic benefits [45]

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10.1530/EJE-10-0859

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