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Furthermore, phenformin was not active in vitro against RSV and IAV

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Notably, treatment with the PKC inhibitor eliminated the difference in adipogenic capacity between control and GPR146-deficient cells, suggesting that impaired PKC signaling underlies the differentiation defect caused by GPR146 loss in both human and mouse preadipocytes (Fig

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The model intracellular concentrations of Hcy, Met, and SAM all fall dramatically corresponding to the clinically observed declines in their plasma concentrations

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