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MNs are miniature, needle-like structures typically ranging in size from hundreds of micrometers to a few millimeters in length

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This is also probably a combination of glucagon and GIP agonism contributing to changes in certain background proteins, specifically lipoprotein lipase, ANGPTL 3 / 8 complexes and ApoC3 as shown in other studies

148,149 Literature shows that altered metabolism in neural cells through aberrant insulin signalling could be a reason for the synthesis of A protein and phosphorylated tau, which are two hallmark biochemical abnormalities linked with AD and proper regulation of insulin signalling has been observed to be a crucial approach in neuroprotection and guarding the synapses against noxious A deposition hence improving cognition in animal models of AD

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