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This study proposed a novel GPCR desensitization mechanism, which differs from the conventional mechanism in which arrestin-induced steric hindrance is a key cellular event

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S-nitrosation of EGFR and src resulted in activation of oncogenic signal transduction pathways, including c-Myc, Akt, and -catenin in human basal-like breast cancer [129]

The constructed pURA3-GSH1 strain was heterozygous at the GSH1 locus, with one allele expressing the URA3 ORF under pGSH1 control and the other expressing GSH1 under pTEF1 control (FigureS1 A )