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[DOI] [PMC free article] [PubMed] [Google Scholar] 149.Fatemeh Y., Siassi F., Rahimi A., Koohdani F., Doostan F., Qorbani M., Sotoudeh G

In cancers, such as esophageal, prostate and cervical cancers, genes such as cyclin dependent kinase inhibitor 2A (CDKN2A, also known as p16INK4a), O-6-methylguanine-DNA methyltransferase (MGMT), retinoic acid receptor (RAR), PC3 and suppressor of cytokine signaling 1 (SOCS1), which were previously hypermethylated, have been demethylated through the action of natural compounds, such as genistein and capsaicin (9)

7a, below) led to a significant decrease in BT549, MDA-MB-231 and Hs578t cell proliferation, which was not related to doxycycline treatment by itself (Supplementary Fig

Brilacidin, a host defense peptide mimetic, potentiates ibrexafungerp antifungal activity against the human pathogenic fungus Aspergillus fumigatus

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