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Treatment with the mitochondrial-targeted antioxidant peptide SS-31 rescues neurovascular coupling responses and cerebrovascular endothelial function and improves cognition in aged mice

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In the hypertrophied and failing heart, decreased myocardial capacity for fatty acid oxidation occurs, leading to enhanced cardiac glucose utilization through anaerobic glycolysis, pyruvate accumulation and lactate production

Lipid depletion led to a dramatic depletion of histone acetylation within 24 hours, and this was rescued by addition of PA/OA (Figure 4E, F

TMEM9 expression was also lower in in normal compared to tumor tissue (Fig

Further, the sample was centrifuged at 5000 g for 5 min at 4C