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Both HCV and HIV infections enhance the induction of oxidative stress

Two possible mechanisms of NO-mediated protein S-glutathionylation can be envisioned: through a GSNO (activated thiol) formation and its consequent reaction with protein-thiol (179) or through an intermediate protein-thiol nitrosylation (activated protein-thiol: analog of sulfenic acid) and its consequent reaction with GSH

B1 (thiamine) deficiency, particularly common in alcohol users, damages peripheral nerves

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